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Description
Dysregulation of mitochondrial oxidative phosphorylation (OXPHOS) complexes was observed in the lungs of DJ-1 knockout mice exposed to aerosolized nicotine, which disrupted the nuclear/mitochondrial stoichiometry resulting in mitochondrial dysfunction
thereby, the CREB1-NRF2 signaling pathway potentiates the function of MSCs, which was shown to increase their therapeutic potential in a humanized mouse model of graft-versus-host disease [37]
Timofeev YS, Kiselev AR, Dzhioeva ON, Drapkina OM
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