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Current research is focused on designing drug candidates that would simultaneously address different mechanisms of high glucose in an individual and contribute to satiation, body weight control and patient convenience

Taken together, these observations indicate a pathogenetic model where intestinal dysbiosis drives subclinical intestinal inflammation followed by disruption of intestinal barrier integrity due to epithelial cell necroptosis and destabilization of TJs (Fig

[DOI] [PubMed] [Google Scholar] 80.Nagamune K, Hicks LM, Fux B, Brossier F, Chini EN, Sibley LD

Loss of intracellular potassium ion is usually accompanied by an increase in intracellular sodium ion