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As the central hub of cellular energy production, mitochondrial damage not only may lead to a decline in ATP synthesis but also may disrupt immune activation and neuronal function through metabolic signaling
As a scaffold protein, the reduction in ABTB1s function will result in the loss of negative regulation of inflammatory pathways such as NF-B through the ubiquitination pathway, leading to excessive activation of inflammatory signals
NO production from solutions of nitrite (10 mM unless otherwise indicated) were stimulated by addition of AA at pH values ranging from 6 to 7
International Journal of Physiology, Pathophysiology and Pharmacology
