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On the other hand, the activation of ACE2 receptors limits activated microglia polarization to the M1 phenotype and shifts its polarization to the M2 anti-inflammatory phenotype [355], which demonstrates the critical role of the renin-angiotensin system (RAS) in neuroinflammation

Furthermore, recent evidence also indicates that the virus may manipulate the mitochondria to maintain its life cycle

Mutations in homocysteine metabolism genes increase keratin N-homocysteinylation and damage in mice

The tight connection between telomeres and mitochondrial function is evident in the activation of p53 when telomere function is impaired, leading to the suppression of PGC1 and PGC1 expression