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A recent trial reported a significant association between elevated LPR during the first 96 h after injury and increased gray matter volume loss at 12 months after TBI (Table 1) [43]

If free radicals are left unchecked, the resulting oxidative stress can lead to: Adequate levels of glutathione can prevent this kind of oxidative stress by neutralizing free radicals

Hence, these thiomethyl metabolites have the potential to be used as diagnostic signals to study susceptibility to hepatotoxicity related to inter-individual production of NAPQI in these types of clinical investigations

Under nutrient-deprived conditions, cancer cells depend on optimal metabolic pathways for survival