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Accordingly, several cellular hallmarks of proteotoxic stress have been identified, including dysregulated mTOR signaling and autophagy, compartment-specific unfolded protein response in mitochondria (UPR mt ), and in the endoplasmic reticulum (UPR ER )

IGF-1 replacement therapy induced a restoration of the altered GH/IGF-1 axis by reducing circulating GH levels and recovering the somatostatinergic tone [292]

Thus, once glucose has been phosphorylated, any potential hindrances until the aldolase step can be bypassed through the pentose phosphate pathway by provision of glyceraldehyde-3-phosphate

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