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confirmation in keratinocyte specific JNK1/2 knockout models will be essential to establish causality unequivocally
doi: 10.1016/j.molstruc.2022.134303
Moreover, metabolic flux analysisusing isotope tracing and computational modelingcan quantify substrate utilization and biosynthetic pathway engagement, providing mechanistic insights into metabolic bottlenecks that limit immune cell fitness or support cancer stem-like cells stemness
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