glutathione content in borrelia Role of metabolism host defense against burgdorferi infection BBE31 from the Lyme disease

GSH regeneration depends on NADPH (pentose phosphate pathway-derived reducing equivalents), critically influencing ferroptotic susceptibility

Sano K, Yosioka I, Kitagawa I

Always read at eye level, using the bottom of the meniscus as your reference point

One should consider the primary phase lesion and hemorrhaging that results from mechanical damage during SCI as well as the secondary phase lesion that lasts several hours or even several months and is accompanied by edema, hemorrhage, inflammation, and cytotoxic edema [44,45,46,47] and may extend to the white matter area and lead to white matter degeneration and damage [48, 49]