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The authors hypothesized that glucose-based components of the dialysate could contribute to the stimulation of eryptosis [265]

This interaction results in the formation of a protein complex that effectively suppresses the ubiquitination and subsequent proteasomal degradation of the transcription factor BACH1, leading to significant stabilization of BACH1 protein levels

W., Hwang, K., & Lipshultz, L

Overall, BPC-157 is thought to act through a coordinated network of biological pathways rather than a single receptor-mediated mechanism
