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Matuszewich L
Pellecchia M, Reduction of circulating cancer cells and metastases in breast-cancer models by a potent EphA2-agonistic peptide-drug conjugate
Directing the nascent protein down the correct PTM pathway prevents accumulation of unfolded species, thought to be the precursor of mutant SOD1 toxicity, premature degradation and aggregation 8,9
Response to growth hormone therapy is generally very good in patients with congenital GH deficiency where a final adult height within parental target range should be expected
