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Description
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Its varied potential mechanisms are thought to include interaction with inducible and/or endothelial nitric oxide-generating systems, by influencing serotonin, dopamine, GABA, and/or opioid metabolism, and/or by directly aiding blood flow, repair, regrowth, angiogenesis, and remodeling
Schmidt, C
BPC-157 is the best characterized at the molecular level with respect to VEGFR2-Akt-eNOS activation and Caveolin-1-mediated endothelial modulation, making it the preferred tool for endothelial cell research [3](#ref-3)
