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These findings are consistent with the primary role of the spleen in the development and progression of MASLD [146]
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tb viability (Venketaraman et al., 2008)
The treatment attenuated VPA-induced behavioral abnormalities in both Y-maze and social play tests, while also restoring prefrontal SOD and GSH-Px activity and normalizing VPA-related changes in neuronal number and soma size (Margedari et al., 2024)
