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Mol Med Rep (2022) 25(1):29
In A42C and S59C, the biotin modification instead takes on a configuration that does not directly block the binding site
To examine the phosphorylation capacities of c-MET mutants, 3UTR siRNA (hs.Ri.MET.13.1 from IDT) was used for selective knockdown of endogenous c-MET in H1299 cell lines that stably express near-endogenous myc-c-MET (WT or mutant)
