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Description
These include experimental models in rats such as acetaminophen hepatitis [36] and hepatic ischaemia/reperfusion injury, where the expression of inflammatory mediators (cyclooxygenase-1 (COX-2), tumour necrosis factor- (TNF-, interleukin (IL)-6, and monocyte chemoattractant protein-1 (MCP-1)) and reactive oxygen species (ROS) production have been demonstrated to be inhibited
4d,e) corpus callosum there were no significant differences between groups
doi: 10.1016/S2213-8587(23)00156-0 84 WysokiskiAStrzeleckiDKoszewskaI
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