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Description
These cell typespecific changes in dopamine receptor expression could explain the lack of significance seen in previous studies that examined PBMCs as a single population and highlights the need for more cell typefocused analyses of dopamine receptor expression and function
Hyperprolinemia type I caused by homozygous p.T466M mutation in PRODH
Gil-Antuano, S
Emerging roles for eicosanoids in renal diseases
