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heteroplasmy and glutathione Schematic illustration of the mechanism by which mitochondrial mtDNA variations in human diseases.

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Other in vitro studies reveal that TNF- could reduce melanocyte-stimulating hormones receptor (MSH-R) binding activity, MC1R expression, and the expression of a melanosomal protein gp87, promoters of melanogenesis 141 , 142

heteroplasmy and glutathione Schematic illustration of the mechanism by which mitochondrial mtDNA variations in human diseases.

10.1371/journal.pone.0245323 156 GudnadottirU.KamauN.FornesR.NguyenM

heteroplasmy and glutathione Schematic illustration of the mechanism by which mitochondrial mtDNA variations in human diseases.

Associations between mercury exposure and the risk of nonalcoholic fatty liver disease (NAFLD) in US adolescents

heteroplasmy and glutathione Schematic illustration of the mechanism by which mitochondrial mtDNA variations in human diseases.

Dark circle improvement effect (4.05%)

heteroplasmy and glutathione Schematic illustration of the mechanism by which mitochondrial mtDNA variations in human diseases.

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