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Concurrently, ATF6 translocates to the Golgi apparatus, where it undergoes proteolytic cleavage to generate cytoplasmic fragment ATF6f, an active transcription factor that modulates the expression of UPR target genes, including XBP1 [10]

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377 In AD mice, injection of BM-MSC-EVs reduced plaque deposition and brain A levels, decreased the activation of astrocytes and microglia, downregulated pro-inflammatory cytokines (TNF- and IL-1), and upregulated anti-inflammatory cytokines (IL-4 and IL-10)