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Chelsea Fox, et.al

By enhancing cholesterol efflux that may in turn be consumed by cholesterol-rich membrane-forming regenerating oligodendrocytes, niacin would prevent prolonged lipid accumulation within macrophages and free up their phagolysosomes for further digestion of engulfed material, while also avoiding a detrimental inflammatory macrophage phenotype (Fig

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The assessment of correlation between these genes and infiltrating immune cells demonstrates that ACO1 and MMD are inversely associated with infiltration of activated mast cells, while ABCC1, CYBB and PEX2 manifest a substantial positive relation with resting mast cells