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Description
In addition to hemodynamic effects, ACEIs/ARBs mitigate fibrotic pathways by suppressing angiotensin II-mediated TGF-1 overexpression in podocytes and tubular cells
Endocrinology 141 , 26082613 (2000)
The topological properties of the drug-disease network were analyzed using Cytoscape software, as demonstrated in Figure 5D
Neurexins, the synaptic adhesion molecules that serve as regulatory centers for synaptic properties, are also the subject of much research on changes in synaptic plasticity and protein levels (Fig
