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Neuroepidemiology 2012;38:117
[2] In the presence of deoxyribonucleic acid (DNA)-damaging factors such as oxidative stress, heat shock, irradiation, and genotoxic drugs potentially leading to oncogene activation, cells stop in their proliferation programs to prevent the passing down of genetic mutations as a tumor suppressive mechanism
Human GMSCs treated with AEDG peptide showed an upregulation of all the genes in question and proteins: Nestin, GAP43, Tubulin III and Doublecortin (Figure 3 and Figure 4)
MG132 treatment further enhanced BPC157-induced BACH1 accumulation (Fig
