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The activation or dysregulation of the complement factors of the alternative complements pathway, like complement factor H (CFH), complement component (C2), complement factor I (CFI), and complement factor B (CFB), have been found to be associated with the development of AMD

MPNSTs are currently untreatable, while only recently an inhibitor of MEK, a downstream effector of Ras signaling, has been approved for pediatric inoperable neurofibromas [2]

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