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Septic cardiomyopathy (SIC) In a model of LPS-mediated septic cardiomyopathy, some researchers revealed that LPS caused ferroptosis and septic cardiac injury by downregulating GPX4 protein levels and increasing COX-2 [93]
Studies have demonstrated that RBP regulates the PI3K/Akt/NF-B signaling pathway to suppress the release of pro-inflammatory cytokines such as TNF-, IL-1, and IL-6
doi: 10.1371/journal.pone.0130993
However, its important to note that GHK-Cu is still primarily used in research environments
