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These pathways include the cyst(e)ine/glutathione (GSH)/glutathione peroxidase 4 (GPX4) axis 1,4 , the ferroptosis suppressor protein 1 (FSP1)/coenzyme Q 10 (CoQ 10 ) axis on the plasma membrane 5,6 , the guanosine triphosphate cyclohydrolase 1 (GCH1)/tetrahydrobiopterin (BH4)/dihydrofolate reductase (DHFR) axis 7,8 , and the mitochondrial dihydroorotate dehydrogenase (DHODH)/CoQ system 9

Part of what makes astaxanthin unique is its ability to cross both the blood-retina and blood-brain barriers, allowing it to reach tissues involved in vision and cognition that many antioxidants cannot readily access.2 Researchers have increasingly explored how astaxanthin works alongside lutein and zeaxanthin, which accumulate in different tissues and have been studied for many of the same outcomes

treatment with 0.5% DSS for 72 h produces marked epithelial disruption, elevates the expression of IL-1, IL-8, and MMP9, and decreases superoxide dismutase (SOD) activity, thereby establishing a model of zebrafish intestinal dysfunction ( Infection-induced models offer unique insights into hostpathogen interactions

Macrophage experiments were conducted by L.B.R.S., X.Q., and B.B., and L.B.R.S