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Activation of redox-sensitive transcription factors further amplifies inflammation by upregulating adhesion molecules and cytokines, facilitating leukocyte infiltration into the endothelium and accelerating vascular complications in the heart, kidneys, and eyes

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The determination of zonulins is not only used to determine the status in the case of "leaky gut"

Its key feature lies in the competitive utilization of the same substrate by iNOS (also known as Nos2) and Arg1: the former generates NO and is closely associated with pro-inflammatory effects, whereas the latter produces ornithine and polyamines and participates in tissue repair and immune regulation ( Under classical pro-inflammatory stimulation (such as LPS/IFN-), iNOS expression is upregulated and catalyzes the conversion of Arg into NO and citrulline