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Dual-pathway interventions that address both sides of the energy equation simultaneously show more sustained effects in observational datasets
Hyperandrogenism can stimulate the secretion of AMH from granulosa cells of over-recruit follicles with the alteration of aromatase activity [27, 28]
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With the increase in diabetes prevalence and mitochondrial toxicity, it is therefore imperative to understand how mitochondrial toxicological agents can potentially compromise insulin sensitivity
