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The integration of these three approaches at the level of the same sample will become an important direction for advancing this field from mechanistic research toward clinical translation (140)

doi: 10.1016/S0015-0282(02)04237-1 109 LiuC-YLeeC-FHongC-HWeiY-H

Silverman MD, Tumuluri RJ, Davis M et al (2002) Homocysteine upregulates vascular cell adhesion molecule-1 expression in cultured human aortic endothelial cells and enhances monocyte adhesion

Specifically, GDH1 deletion led to decreased binding of Sir2 protein on the telomeres, causing elevated transcript levels of genes affected by the loss of the SIR complex [40]