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DFO upregulates GPX4, xCT, and GSH, curbs gliocyte overgrowth, enhances long-term motor function, and mitigates iron overload (Yao et al., 2019)
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doi:10.1016/j.clinbiochem.2021.04.023
Ribonucleosides (adenosine, guanosine, inosine) are first degraded to their respective bases (adenine, guanine, hypoxanthine) by purine nucleoside phosphorylase (PNP), then salvaged to NMPs via PRPP-dependent enzymes adenine phosphoribosyltransferase (APRT) and hypoxanthineguanine phosphoribosyltransferase (HGPRT) [37, 38]
