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Other mechanisms for controlling cellular abundance of iron influence the sensitivity of cells to ferroptosis: iron export through ferroportin, or through prominin2-mediated ferritin-containing multivesicular bodies (MVBs) and exosomes drives resistance to ferroptosis (Brown et al., 2019), as this depletes the cellular pool of iron, and its ability to promote lipid peroxidation

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There, CamKII kinase activity enhances AMPAR-mediated transmission in two ways

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Experimental evidence supporting a role for nicotine includes its potential capacity to inhibit the action of striatal DAT or to increase action potentials along nigrostriatal axons, increasing dopamine levels in the synaptic gap 67,68

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