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FTIR spectroscopy validated CAY10603 loading and lactoferrin conjugation, while the EE% was determined to be 20.57 1.21% with DL% of 0.69 0.06%

It also modulates nitric oxide pathways and growth factor signaling, supporting gut mucosal integrity and reducing systemic inflammation

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This suggests that the co-treatment with ferrous iron again switched the signaling mechanisms and mode of cell death from mitochondrial dysfunction-dependent necrosis after APAP alone to LPO-dominated ferroptosis after APAP + Fe 2+ (Figure 1)