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Description
Traditional oral pills often achieve less than 20% absorption
It has been shown that IGFR is absolutely necessary to induce PI3K/Akt pathway and differentiate granulosa cells by FSH [20, 21, 24, 25]
Although their role in CIPN has been known for a long time, there are limited reports about the interaction between these factors and TRP channels in the literature
Growth hormone stimulates hepatic IGF-1 production by binding to growth hormone receptors on liver cells, triggering intracellular signaling through the JAK2/STAT5 pathway that upregulates IGF-1 gene expression
