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Notably, interleukin-1 binds the IL-1 receptor type I and induces enzymes that degrade the extracellular matrix, including matrix metalloproteinases (MMPs) and a disintegrin and metalloproteinase with thrombospondin motifs (ADAMTS), thereby amplifying synovitis and initiating progressive cartilage loss (Wang et al., 2024)
Fusco, R
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A range of drugs directed towards the treatment of IC have been the topic of recent debate, although the largest percentage of patients cured of the condition remains in the region of 60% [5]
