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The IL-17 secreted by these cells binds to IL-17 receptors (IL-17Rs) expressed in midbrain neurons, inducing neuronal death through the upregulation of NF-B and downstream signaling pathways, as demonstrated in human iPSC-derived neuronal models
Neuronal expression of EAAT1 is correlated with neurofibrillary tangle formation (Scott et al., 2002), while glutamine synthetase expression corresponds with plaque formation (Robinson, 2000)
