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Inhalation of PGE2 in the form of liposomes into the lung can effectively improve pulmonary fibrosis, which is expected to become a therapeutic option for inhaled treatment of pulmonary fibrosis (Ivanova et al., 2013)

Given that glutaryllipoyl adducts form under conditions of glutarate excess on another TCA cycle ketoacid dehydrogenase, the PDHc 9 , we hypothesized that the major role of ABHD11 may be to reverse lipoyl glutarylation, maintaining TCA cycle integrity (Fig

At the same time, it should be acknowledged that IL-33 can also amplify type-2 responses in other contexts, underscoring the importance of dose, timing, and tissue state in determining its net effect (Wilson, 2020

Several enzymes that may be related to EPS degradation were also detected in the metagenome of the DW biofilm (Fig