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These transport systems create spatial regulation of metal-induced oxidative damageCus extracellular antioxidant role via SOD3 contrasts with its intracellular mitochondrial toxicity (75, 76), while Zns synaptic release versus cytoplasmic accumulation dictates its dual neuroprotective/pro-oxidant effects (69, 74)

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These dynamic and heterogeneous shifts in cell death modalities across disease stages and brain regions significantly complicate PD treatment, necessitating in-depth research to develop targeted therapeutic strategies