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Environmental exposures, including pesticides (rotenone, paraquat), heavy metals (manganese, mercury, lead), and air pollutants, significantly increase PD risk through oxidative mechanisms and inflammatory pathways [27]

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Cruciferous vegetable consumption alters the metabolism of the dietary carcinogen 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in humans

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