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This coordinated targeting strategy significantly amplified antitumor efficacy by reshaping glucose-dependent immune suppression, while maintaining favorable systemic safety, as validated in both tumor-bearing mice and non-human primates

Large-scale postmortem analyses indicate that cerebrovascular pathology occurs in approximately 80% of AD casesa prevalence markedly higher than in other neurodegenerative disorders such as Parkinsons disease, frontotemporal dementia, and Lewy body diseasehighlighting a potentially distinctive role of vascular dysfunction in AD pathogenesis ( Astrocytes, the predominant glial cell type in the brain, are strategically positioned at the BBB interface and contribute critically to cerebral homeostasis, neuronal support, and immune modulation ( Although astrocyte dysfunction and iron dysregulation are increasingly recognized in AD pathology, their direct causal link to disease progressionand particularly to hypothesized central mechanisms such as ferroptosisrequires further validation in human contexts

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