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However, in vivo , the mechanisms driving this M1 to M2 phenotypic switch may be more complicated and may be regulated by IL-10, glucocorticoids, prostaglandins, miRNAs, and adenosine and peroxisome proliferator-activated receptor (PPAR-) signaling in the wound microenvironment (8688, 109)

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doi:10.1017/S0007114509359127 Hanna M, Jaqua E, Nguyen V, Clay J