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Zeng Q, Xu J, Mu X, et al

The proposed mechanism for hyperhomocysteinaemia on inducing endothelial dysfunction and thus atherosclerosis includes homocysteine-induced endoplasmic reticulum stress, oxidative stress, and proinflammatory response.48 Animal models of hyperhomocysteinaemia have confirmed the causal relationship between hyperhomocysteinaemia and the development of endothelial dysfunction and accelerated atherosclerosis.48 Although meta-analyses have shown reduction of cardiovascular risk with reduction of homocysteine levels,46 47 vitamin supplementation (with vitamin B 6 , vitamin B 12 , and folic acid) to lower homocysteine in the body may not be transformed into clinically beneficial vascular outcomes

The report singled out the San Diego-based Boxer Amphibious Ready group, which was composed of the USS Boxer, the USS Somerset and the USS Harpers Ferry, which had been scheduled to deploy in September 2023

It does not appear on the Drug Product Database as an approved therapeutic