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A prior study suggested that mutation of six distinct serine or threonine residues located throughout the C-terminal half of ArfGAP1 completely prevented its phosphorylation by LRRK2, but the relative contribution of phosphorylation at each site was not clearly determined (Xiong et al., 2012)

doi: 10.3389/fnagi.2025.1535280 Received 27 November 2024 Accepted 13 January 2025 Published 29 January 2025 Volume 17 - 2025 Edited by Wei-Jye Lin, Sun Yat-sen Memorial Hospital, China Reviewed by Celia Giulietta Fernandez, Recursion Pharmaceuticals, United States Barbara Stopschinski, University of Texas Southwestern Medical Center, United States Updates Copyright 2025 Islam, Noorani, Sun, Michikawa and Zou

This phenomenon could potentially be ascribed to the downregulation of NF-B activity by specifically targeting SHIP1 in microglia, offering a hopeful strategy for safeguarding neurological health ( 5.19 miR-335 The levels of miR-335 expression showed significant decrease in different PD mimic scenarios, including the stimulation of LPS in BV-2 microglia and the upregulation of wild-type LRRK2