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The residual cells were arrested at the G0/G1 phase of the cell cycle, and the percentage of cells in the S phase was reduced, while the parental and regrown cells showed the similar cell cycle distribution

When mitochondria function optimally, you have: High energy levels Efficient fat burning Good insulin sensitivity Low inflammation Healthy cellular function When mitochondria become dysfunctional through aging, poor diet, toxins, stress, or chronic illness you develop: Metabolic inflexibility (inability to switch between burning carbs and fat) Insulin resistance Reduced energy production Increased oxidative stress Accelerated aging Chronic disease This is where MOTS-C becomes revolutionary

These aldehydes modify critical proteins via adduct formation, alter intracellular signaling, and promote the activation of NF-B and other pro-inflammatory pathways [34,35], which exacerbate local inflammation and contribute to the systemic oxidative burden observed in psoriatic patients [36]