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HSP90 inhibitors such as ganetespib (STA-9090) and AUY922 have been extensively evaluated in oncology, 54,55 where they disrupt the stabilization of oncogenic client proteins

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This occurs as a consequence of a depletion of the hepatic levels of glutathione due to the sustained plasmatic and biliary exportation from the cell as GSSG to maintain the GSH/ GSSG ratio.83 The above-mentioned mechanisms may be predominant under strong oxidizing conditions