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Several studies indicate that human cells can also actively trigger ROS production at small doses, as part of signaling pathways, regulating cell survival and proliferation, as a defense mechanism against invaders (Sena and Chandel, 2012)
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Because they rely on the standard digestive process, it can take much longer to reach a "steady state" in the blood
This link establishes a direct connection between the oxidative inflammatory microenvironment and the central mechanisms underlying RA pathophysiology
