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This is because Mfn2 overexpression can mediate smooth muscle cell apoptosis by inhibiting protein kinase B (Akt) signaling and activating mitochondrial apoptosis (185)
10.3390/cancers13020188 26 CabasagC
Macrophages differentiated under hypoxic conditions exhibit diminished supersulfide levels and display an exaggerated and prolonged inflammatory response to LPS stimulation, attributed to the loss of TET2 function, a key mediator of inflammation resolution (see Fig
