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Pharm Res 32:121
In Parkinsons disease, curcumin inhibits DNMTs, leading to demethylation and reactivation of neuroprotective genes like Nurr1, DJ-1, and PARK genes, and acts as an HDAC inhibitor to enhance histone acetylation, promoting transcription of genes involved in neuronal survival and synaptic plasticity
Moreover, this implies that glycogen can be converted into other energy substrates, such as glucose and lactate, whenever necessary for maintaining brain functionality
