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Description
In chronic or biofilm-associated infections, however, microbial persistence and metabolic cues from the wound microbiome can skew phagocyte polarization toward a dysfunctional state either sustaining a prolonged M1-like inflammatory profile that damages tissue or inducing an immunosuppressed M2-like state that allows microbial survival [42]
Hormone signaling Local estrogen production may play a pathophysiological role in endometriosis by enhancing the inflammatory status of the endometrium [81]
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Dupoiron D, Richard H, Chabert-Desnot V et al
