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105 OATPs and Rotor syndrome [Mendelian inheritance in man237450] Rotor syndrome is an autosomal recessive disorder characterized by mild conjugated and unconjugated hyperbilirubinemia caused by inactivating mutations or deletion of both SLCO1B1 and SLCO1B3 , which encode OATP1B1 and OATP1B3, respectively [Tables 2 & 3]

Recent studies have demonstrated that FABPs selectively cooperate with the nuclear receptors RAR, ERR 47 , PPAR/, and PPAR

The hepatotoxicity and synergy between the two substances were analyzed using quantitative data (cell viability), as well as qualitative data (cell morphology and histologic analysis)