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Mitochondrial dysfunction in cardiovascular diseases: potential targets for treatment

Glutatyon eksiklii yaanmas, hcrelerin zararl serbest radikaller ve toksinlerle mcadele etme yeteneini azaltarak baklk sisteminin zayflamasna ve hcre hasarnn artmasna yol aabilir

and ribosomal protein production machinery, as outlined in several proteome resource studies [2, 5,6,7]

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