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Clinical studies indicate that elevated oxidative stress markers persist into the subacute and chronic phases of stroke, potentially limiting neuroplasticity and functional recovery (42)
Although it remains controversial whether ferroptosis is the ultimate causative factor for the condition, the overexpression of GPX4 prevents I/R damage in certain tissues by limiting lipid peroxidation [189]
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10.1002/pros.20690 19 ChoH.-J.AhnK.-C.ChoiJ
