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Description
This ROS-mediated neuronal damage occurs due to the disruption of redox reaction, i.e., decrease in the activities of antioxidant enzymes such as superoxide dismutase and catalase activities, and a decrease in the level of antioxidants (ascorbic acid and tocopherol) favors the surplus ROS production along with increased accumulation of A in AD patients [119]

PTEN, gene of phosphate and tensin homolog deleted on chromosome ten

The effect of a leukotriene B4 antagonist LY293111 on allergen- induced responses in asthma
Felix, I
