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Description
Nevertheless, experimental data suggest that the altered intracellular redox status associated with hyperhomo-cysteinemia-induced oxidant stress is the major molecular mechanism responsible for the atherothrombogenicity of moderately elevated circulating homocysteine
doi: 10.3109/0886022X.2011.618972
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It promotes angiogenesis (the formation of new blood vessels), boosts collagen production, and reduces inflammation
